Материал: Донсков С.И., Мороков В.А. Группы крови человека. Руководство по иммуносерологии

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103.McGinnis M.H., Macher A.M., Rook A.H., Alter H.J. Red cell autoantibodies in patients with acquired immune deficiency syndrome // Transfusion. – 1986. – V. 26. – P. 405–409.

104.Mollison P.L., Engelfriet P., Contreras M. Blood Transfusion in Clinical Medicine. –10-th ed. – Oxford: BSP, 1997. – 1033 p.

105.Morel P., Garratty G., Willbanks E. Another example of anti-IB // Vox Sang. – 1975. –

V.29. – P. 231–233.

106.Navenot J.-M., Muller J.-Y., Blanchard D. Expression of blood group i antigen and fetal hemoglobin in paroxysmal nocturnal hemoglobinuria // Transfusion. – 1997. – V. 37. –

P.291–297.

107.Niemann H., Watanabe K., Hakomori S.-I. et al. Blood group i and I activities of ‘lacto-N- norhexaosylceramide‘ and its analogues: the structural requirements for i-specificities // Biochem. Biophys. Res. Commun. – 1978. – V. 81. – P. 1286–1293.

108.OgataH.,OkuboY.,AkabaneT.PhenotypeiassociatedwithcongenitalcataractinJapanese // Transfusion. – 1979. – V. 19. – P. 166–168.

109.Okada Y., Kannagi R., Levery S.B., Hakomori S.-I. Glycolipid antigens with blood group I and i specificities from human adult and umbilical cord erythrocytes // J. Immunol. – 1984. – V. 133. – P. 835–842.

110.Okubo Y., Yamaguchi H. I-negative phenotype and cataract [Abstract] // 19-th Cong. Int. Soc. Blood Transfus., 1986. – P.147.

111.Olesen H. Thermodynamics of the cold agglutinin reaction // Scand. J. Clin. Lab. Invest. – 1966. – V. 18. – P. 1–15.

112.Page P.L., Langevin S., Petersen R.A., Kruskall M.S. Reduced association between Ii blood group and congenital cataract in white patients // Amer. J. Clin. Path. – 1987. – V. 87. –

P.101–102.

113.Pascual V., Victor K., Lelsz D. et al. Nucleotide sequence analysis of the V regions of two

IgM cold agglutinins: evidence that the VH4-21 gene segment is responsible for the major cross-reactive idiotype // J. Immunol. –1991. – V. 146. – P. 4385–42391.

114.Pawlak Z., Lopez M. Development des antigenesABH et Ii chez de 0 a 16 ans // Rev. Franc. Transfus. Immunohemat. – 1979. – V. 22. – P. 253–263.

115.Pereira A., Mazzara R., Escoda I. et al. Anti-Sa cold agglutinin of IgA class requiring plasma exchange-therapyasearlymanifestationofmultiplemyeloma//Ann.Hematol.–1993.–V.66.–

P.315–318.

116.Picard J., Edward D.W., Feizi T. Changes in expression of the blood groupA, B, H, Le a and Le b antigens and the blood group precursors associated I (MA) antigen in glycoprotein-rich extracts of gastric carcinomas // J. Clin. Lab. Immunol. – 1983. – V. 1. – P. 119–128.

117.Picard J., Feizi T. Peanut lectin and anti-Ii antibodies reveal structural differences among human gastrointestinal glycoproteins // Mol. Immunol. – 1983. – V. 20. – P. 1215–1220.

118.Picard J.K., Loveday D., Feizi T. Evidence for sialylated Type I blood group chains on

human erythrocyte membranes revealed by agglutination of neuraminidase-treated erythrocytes with Walenstrom’s macroglobulin IgM WOO and hybridoma antibody FC 10.2 // Vox Sang. – 1985. – V. 48. – P. 26–33.

119.Piller F., Cartron J.-P., Maranduba A. et al. Biosynthesis of blood group I antigens. Identification of a UDP-GlcNAc:GlcNAcβ1-3Gal(-R)β1-6(GlcNAc to Gal) N-acetylglucosaminyltransferase in hog gastric mucosa // J. Biol. Chem. – 1984. –V. 259. –

P.13385–13390.

120.Postoway N., Capon S., Smith L. еt al. Cold agglutinin syndrome caused by anti-I T [Abstract] // Joint. Cong. Int. Soc. Blood Transfus. аndAABB, 1990. – P. 85.

121.Pruzanski W., Delmage K.J. Cytoxic and cytolytic activity of homogeneous cold agglutinins ofperipheralbloodmonocytes//Clin.Immunol.Immunopathol.–1977.–V.7.–P.130–138.

122.PruzanskiW.,FaridN.,KeystoneE.etal.Theinfluenceofhomogeneouscoldagglutininson humanBandTlymphocytes// Clin.Immunol. Immunopathol.–1975.–V. 4. –P. 248–257.

891

123.Pruzanski W., Farid N., Keystone E., Armstrong M. The influence of homogenous cold agglutinins on polymorphonuclear and mononuclear phagocytes // Clin. Immunol. Immunopathol. – 1975. – V. 4. – P. 277–285.

124.Pruzanski W., Shumak K.H. Biologic activity of cold-reacting agglutinins // N. Engl.

J.Med. – 1977. – V. 297. – P. 538–542.

125.Pruzanski W., Shumak K.H. Biologic activity of cold-reacting autoantibodies // N. Engl.

J.Med. – 1977. – V. 297. – P. 583–589.

126.Race R.R., Sanger R. Blood Groups in Man. – 6-th ed. – Oxford: BSP, 1975. – 659 p.

127.Ramos R.R., Curtis B.R., Eby C.S. et al. Fatal outcome in a patient with autoimmune hemolytic anemia associated with an IgM bithermic anti-I TP // Transfusion. – 1994. –

V.34. – P. 427–431.

128.Reid M.E., Bird G.W.G. Association between human red cell blood group antigens and disease // Transfus. Med. Rev. – 1990. – V. 4. – P. 47–55.

129.Reid M.E., Lomas-Francis C. The Blood GroupAntigen: FactsBook. – 2-nd ed. – London: Academic Press, 2004. – 561 p.

130.Roelcke D.Afurther cold agglutinin, Fl, recognizing a N-acetylneuraminic acid-determined antigen // Vox Sang. – 1981. – V. 41. – P. 98–101.

131.Roelcke D. Cold agglutination // Transfus. Med. Rev. – 1989. – V. 3. – P. 140–146.

132.Roelcke D. Li cold agglutinin: a further antibody recognizing sialic acid-dependent antigen fully expressed on newborn erythrocytes // Vox Sang. – 1985. – V. 48. – P. 181–183.

133.Roelcke D. Sialic acid-dependent red blood cells antigens // G.Garratty, ed. Immunobiology of Transfusion Medicine. – NY: Dekker, 1994. – P. 69–95.

134.Roelcke D.The Lud cold agglutinin: a further antibody recognizing N-acetylneuraminic aciddetermined antigens not fully expressed at birth //Vox Sang. – 1981. –V. 41. – P. 316–318.

135.Roelcke D., Brossmer R. Different fine specificities of human monoclonal anti-Gd cold agglutinins // Prot. Biol. Fluids. – 1984. – V. 31. – P. 1075–1078.

136.Roelcke D., Hack H., Kreft H. et al. IgA cold agglutinins recognize Pr and Sa antigens expressed on glycophorins // Transfusion. – 1993. – V. 33. – P. 472–475.

137.Roelcke D., Hack H., Kreft H., Gross H.J. α2,3-Specific desialylation of human red cells: effect on the autoantigens of the Pr, Sa and Sia-I1, b1, Ib1 series // Vox Sang. – 1998. –

V.74. – P. 109–112.

138.RoelckeD.,HenggeU.,KirschfinkM.Neolasto(Type-2chain)-sialoautoantigensrecognized by human cold agglutinins // Vox Sang. – 1990. – V. 59. – P. 235–239.

139.Roelcke D., Kreft H., Hack H., Stevenson F.K. Anti-i: human cold agglutinins recognizing linear (i) and branched (I) Type 2 chains // Vox Sang. – 1994. – V. 67. – P. 216–221.

140.Roelcke D., Kreft H., Northoff H., Gallasch E. Sia-b1 and I antigens recognized by

Mycoplasmapneumoniae-inducedhumancoldagglutinins.//Transfusion.–1991.–V.31.– P.627-630.

141.RoelckeD.,KreftH.,PfisterA.-M.ColdagglutininVo:anIgMλmonoclonalhumanantibody recognizingasialicaciddeterminedantigenfullyexpressedonnewbornerythrocytes//Vox Sang. – 1984. – V. 47. – P. 236–241.

142.Roelcke D., Pruzanski W., Ebert W. et al. A new human monoclonal cold agglutinin Sa recognizing terminal N-acetylneuraminyl groups on the cell surface // Blood. – 1980. –

V.55. – P. 677–681.

143.Roelcke D., Riesen W., Geisen H.P., Ebert W. Serologial identification of the new cold agglutinin specificity anti-Gd // Vox Sang. – 1977. – V. 33. – P. 304–306.

144.Rosenfield R.E., Schmidt P.J., Calvo R., McGinnis M.H.Anti-i, a frequent cold agglutinin in infectious mononucleosis //Vox Sang. – 1965. – V. 10. – P.631-634.

145.Rouger P., Juszczak G., Doinel C. et al. Relationship between I and H antigens. II. Study of the H and I deficient phenotypes // Immunol. Commun. – 1980. – V. 9. – P. 161–172.

892

146.Rouger P., Juszczak G., Doinel C., Salmon C. Relationship between I and H antigens.

I.Astudy of the plasma and saliva of a normal population // Transfusion. – 1980. – V. 20. –

P.536–539.

147.Rouger P., Riveau D., Salmon C. Detection of the H and I blood group antigens in normal plasma: a comprasion withAand i antigens // Vox Sang. – 1979. – V. 37. – P. 78–83.

148.Salama A., Pralle H., Mueller-Eckhardt C. A new red blood cell cold autoantibody (antiMe) // Vox Sang. – 1985. – V. 49. – P. 277–284.

149.Salmon C., Homberg J.C., Liberge G., Delarue F.Autoanticorps a specificieties multiplex, anti-HI, antiAI, anti-BI, dans certains eluats d’anemie hemolytique // Rev. Franc. Etud. Clin. Biol. – 1965. –V. 10. – P. 522–525.

150.Schmidt P.J., McCurdy P., Havell T. еt al. An anti-I T of clinical significance [Abstract] // Transfusion. – 1974. –V. 14. – P. 507.

151.Schutte M.E.M., van Es J.H., Silbersten L.E., Logtenberg T. VH4.21-encoded natural autoantibodies with anti-i specificity mirror those associated with cold hemagglutinin disease // J. Immunol. – 1993. – V. 151. – P. 6569–6576.

152.Shirey R.S., Park K., Ness P.M. et al. An anti-i biphasic hemolysin in chronic paroxysmal cold hemoglobinuria // Transfusion. – 1986. – V. 26. – P. 62–64.

153.Shumak K.H., Baker M.A., Taub R.N. et al. Myeloblastic and lymphoblastic markers in acute undifferentiated leukemia and chronic myelogenous leukemia in blast crisis // Cancer Res. – 1980. – V. 40. – P. 4048–4052.

154.Shumak K.H., Beldotti I., Rachkewich R.A. Diagnosis of haematological disease using anti-i. I. Disorders with lymphocytosis // Brit. J. Haemat. – 1979. – V. 41. – P. 399–405.

155.Shumak K.H., Rachkewich R.A., Beldotti I. Diagnosis of haematological disease using anti-i.II.Distinctionbetweenacutemyeloblasticandacutelymphoblasticleukaemia//Brit.

J.Haemat. – 1979. – V. 41. – P. 407–411.

156.Shumak K.H., Rachkewich R.A., Crookston M.C., Crookston J.H.Antigens of the Ii system on lymphocytes // Nature New Biol. – 1971. – V. 231. – P. 148–149.

157.Shumak K.H., Rachkewich R.A., Greaves M.F. I and i antigens on normal human T and B lymphocytes and on lymphocytes from patient with chronic lymphocytic leukemia // Clin. Immunol. Immunopathol. – 1975. – V. 4. – P. 241–247.

158.Signal T., Booth P.B.ANew Zealand family with i members // Vox Sang. – 1976. – V. 30. –

P.391–395.

159.Silberstein L.E., Jefferies L.C., Goldman J. et al. Variable region gene of pathologic human autoantibodiestotherelatediandIredbloodcellantigens//Blood.–1991.–V.78.–P.2372– 2386.

160.SilvergleidA.J., Wells R.F., Hafleigh E.B. et al. Compability test using 51Chromium-labeled red blood cells in cross-match positive patients // Transfusion. – 1978. – V. 18. – P. 8–14.

161.Smith G., Spellerberg M., Boulton F. et al. The immunoglobulin VH gene, VH4-21, specificallyencodesautoanti-redantibodiesagainsttheIoriantigens//VoxSang.–1995.–

V.68. – P. 231–235.

162.Stevenson F.K., Smith G.J., North J. et al. Identification of normal B-cell counterparts of neoplastic cells which severe cold agglutinins of anti-I and anti-i specificity // Brit.

J.Haemat. – 1989. – V. 72. – P. 9–15.

163.Stevenson F.K., Wrightham M., Glennie M.J. et al.Antibodies no shared idiotypes as agents for analysis and therapy for human B cell tumors // Blood. – 1986. – V. 68. – P. 430–436.

164.Tegoli J., Cortez M., Jensen L., Marsh W.L. A new antibody, anti-ILe bH, specific for a determinant formed by the combined action of I, Le, Se and H gene products //Vox Sang. – 1971. – V. 21. – P. 397–404.

165.Tegoli J., Harrris J.P., Issitt P.D., Sanders C.W. Anti-IB, an expected ‘new’ antibody detecting a joint product of the I and B genes // Vox Sang. – 1967. – V. 13. – P. 144–157.

893

166.Terness P., Navolan D., Opelz G., Roelcke D. Inverse association between IgG-anti-κ and antierythrocyte autoantibodies in patients with cold agglutination // Blood. – 1999. –

V.94. – P. 4343–4346.

167.Testa U., Henri A., Bettaieb A. et al. Regulation of i- and I-antigen expression in the K562 cell line // Canser Res. – 1982. – V. 42. – P. 4694–4700.

168.Testa U., Rochant H., Henri A. et al. Change in i-antigen expression of erythrocyte during in vivo aging // Rev. Franc. Transfus. Immunohemat. – 1981. – V. 24. – P. 299–305.

169.ThomasD.B.Theiantigencomplex:anewspecificityuniquetodividinghumancells//Eur.

J.Immunol. – 1974. – V. 4. – P. 819–824.

170.Thorpe S.J., Boult C.E., Stevenson F.K. et al. Cold agglutinin activity is common among

human monoclonal IgM Rh system antibodies using theV4-34 heavy chain variable segment // Transfusion. – 1997. – V. 37. – P. 1111–1116.

171.Thorpe S.J., Turner C.E., Stevenson F.K. et al. Human monoclonal antibodies encoded by the V4-34genesegmentshowcoldagglutininactivityandvariablemultireactivitywhichcorrelates with the predicted charge of the heavy-chain variable region // Immunology. –1988. – V. 93. –

P.129–136.

172.Tippett P., Noades J., Sanger R. et al. Further studies of the I antigen and antibody // Vox Sang. – 1960. – V. 5. – P. 107–121.

173.Troxel D.B., Innella F., Cohen R.J. Infectious mononucleosis complicated by hemolytic anemia due to anti-i //Amer. J. Clin. Path. – 1966. – V. 46. – P. 625–631.

174.Uemura K., Roelcke D., Nagai Y., Feizi T. The reactivities of human erythrocyte

autoantibodies anti-Pr2, anti-Gd, Fl and Sa with gangliosides in a chromatogram binding assay // Biochem. J. – 1984. – V. 219. – P. 865–874.

175.Van Loghem J.J., van der Hart M., Veenhoven-van Riesz E. et al. Cold auto-agglutinins and haemolysins of anti-I and anti-i specificity // Vox Sang. – 1962. – V. 7. – P. 214–221.

176.Watanabe K., Hakomori S., Chids R.A., Feizi T. Characterization of a blood group I-active ganglioside: structural requirements for I and i specificities // J. Biol. Chem. – 1979. –

V.254. – P. 3221–3228.

177.Watanabe K., Hakomori S.-I. Status of blood group carbohydrate chains in ontogenesis and in oncogenesis // J. Exp. Med. – 1976. – V. 144. – P. 644–653.

178.Weiner W., Shinton N.K., Gray I.R. Antibody of blood-group specificity in simple (‘cold’) haemolytic anaemias // J. Clin. Pathol. – 1960. – V. 13. – P. 232–236.

179.Wiener A.S., Moor-Jankowski J., Gordon E.B., Davis J. The blood group factors I and i in primates including man, and in lower species // Amer. J. Phys. Anthrop. – 1965. – V. 23. –

P.389–396.

180.WienerA.S.,UngerL.J.,CohenL.,FeldmanJ.Type-specificcoldauto-antibodiesasacause ofacquiredhemolyticanemiaandhemolytictransfusionreactions:biologictestwithbovine red cells //Ann. Intern. Med. – 1956. – V. 44. – P. 221–240.

181.WilkinsonL.S.,PetzL.D.,GarrattyG.Reappraisaloftheroleofanti-iinhaemolyticanemia in infectious mononucleosis // Bitr. J. Haemat. – 1973. – V. 25. – P. 715–722.

182.Winkler M.M., Hamilton J.R. Previously tested donors eliminated to determine rare phenotype frequencies [Abstract] // Joint. Congr. Int. Soc. Blood Transfus. and AABB, 1990. – P.158.

183.Yamaguchi H., Okubo Y., Tanaka M.Anote on possible close linkage between the Ii blood locus and a congenital cataract locus // Proc. Jpn.Acad. – 1972. – V. 48. – P. 625–628.

184.Yamaguchi H., Okubo Y., Tomita T. et al. A rare I (I-negative) phenotype blood found in Japanese families // Proc. Jpn.Acad. – 1970. – V. 46. – P. 889–892.

185.Yu L.-C., Twu Y.-C., Chang C.-Y., Lin M. Molecular basis of the adult i and the gene responsible for the expression of the human blood group I antigen // Blood. – 2001. –

V.98. – P. 3840–3845.

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Глава 28.

Коллекция 208 (Er)

Кколлекции 208 отнесены три антигена: 2 часто встречающихся (Er a и Er3)

и1 (Er b), встречающийся крайне редко (табл. 28.1). Известен нулевой фенотип – Er(a −b −). Антигены Er a и Er b – антитетичны. Коллекция Er не получила статус групповой антигенной системы, поскольку не установлена хромосомная локализация генов ER.

Таблица 28.1

Антигены Er

Обозначение

 

Частота, %

традиционное

ISBT

 

Er a

ER1

 

208001

> 99,9

Er b

ER2

 

208002

< 0,01

Er3

ER3

 

208003

> 99,9

Серология антигенов Er

Антиген Er a описали Daniels и соавт. [2] в 1982 г., обнаружив у одного из пациентов не идентифицированные ранее антитела. Антитела такой же специфичности находили и другие исследователи. В соответствии с их обозначениями антиген Er a

именовалсякакRosebush,Ros,MinиRod(Reid,Lomas-Francis[9).Сравнениеанти-

телпоказало,чтоониреагируютсоднимитемжеантигеном–Er a.

В 1988 г. Hamilton и соавт. [5] нашли у женщины, имевшей пять беременностей, антитела с очень низкой частотой реагирования. Они взаимодействовали с пятью из шести образцов эритроцитов Er(a −). Открываемый ими антиген получил обозначение Er b.

Интересна семья Rod., в которой были обнаружены анти-Er b-антитела. В двух поколениях этой семьи были лица Er(a +b + ), Er(a −b + ) и Er(a −b −), и это позволило Hamilton и соавт. показать, что гены Er a и Er b наследуются кодоминантно, а редкий нулевой фенотип является следствием гомозиготности по молчащему аллелю ER, также передаваемому по наследству в соответствии с законом Менделя.

Следует упомянуть еще одну работу, дополняющую серологическую характеристикуколлекции208.Arriagaисоавт.[1]обнаружилииндивидаEr(a −b −),сыворотка крови которого реагировала с эритроцитами как Er(a −b + ), так и Er(a +b −). Антитела не удавалось разделить путем дифференциальной адсорбции, в связи с

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